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SCHIZOPHRENIA Presentation Transcript:
1.SCHIZOPHRENIA
2.Epidermology
1 % of Adults
A mean Duration – 15 Years
Peak Incidence 25-30yrs
Smaller Peak – Over 65
High Rates – Lower Social Classes
Central zones of larger cities
3.Clinical Features
Syndrome of abnormalities
Several Spheres
4.Symptoms
5.Type 1 Schizophrenia
Type 2 Schizophrenia?
Acute undifferentiated Schizophrenia
Paranoid Schizophrenia
Chronic schizophrenia
6.Schizophrenia Or Mania?
Differentiation?
Mental State
?Mania-Pronounce elation of mood
Course
?Repeated episodes occur in
both.
?Gradual accumulation of (-) Symp.
7.Symptomatic Schizophrenia Amphetamione psychosis
Alcoholic hallucinosis
Auditory Hallucinations (primary)
Dellusions(secondary)
Toxic Psychosis due to hallucinogens
Mescaline, lysergic acid and psilocylin
Differentiable in color and shape constancy.
Paranoid psychosis
Delusions
Development of sensitive, suspicious and rigid personality.
Jealousy and erotomania
Cycloid Psychoses
Manic –depressive Illness
The excited pole and a inhibited depressesed pole
8. Amphetamines and cocaine
L-Dopa -increases the levels of dopamine
Neuroleptic drugs (chlorpromazine) ?Immediately on Hallucinations
?Weeks to affect the behaviour of the patients. Dopamine hypothesis is unable to explain this delay.
Chlorpromazine?little/no difference to 30%
9.Chlorpromazine - an anti-psychotic drug
Clozapine – To treat the Negative Symptoms
Both dopamine and serotonin are
probably involved, but the precise role played by each is unclear.
10.Acute Schizophrenia ( Type 1) + symptoms
? May make Complete recovery.
Chronic Schizophrenia
?Patient progressively may get disabled
10% - Rapid & Permanent Deterioration
35% - Mild & Persistent Symptoms
35% - Appear Cure but have relapse of Illness
20% - Apparently cure and stable
11.Physical
Drug Screening
Test for organic illness
Psycho- Social
History
Asses Home environment
12.Acute Phase
Long term Treatment
13.Pre Morbid:
Family History
Schizoid Personality
Home environment
Poor Previous Work Record
Illness
Gradual Onset
Absence of Prominent Affective Symptoms
Pronounced Negative Symptoms
SCHIZOPHRENIA Presentation Transcript:
1.SCHIZOPHRENIA
2.Epidermology
1 % of Adults
A mean Duration – 15 Years
Peak Incidence 25-30yrs
Smaller Peak – Over 65
High Rates – Lower Social Classes
Central zones of larger cities
3.Clinical Features
Syndrome of abnormalities
Several Spheres
4.Symptoms
5.Type 1 Schizophrenia
Type 2 Schizophrenia?
Acute undifferentiated Schizophrenia
Paranoid Schizophrenia
Chronic schizophrenia
6.Schizophrenia Or Mania?
Differentiation?
Mental State
?Mania-Pronounce elation of mood
Course
?Repeated episodes occur in
both.
?Gradual accumulation of (-) Symp.
7.Symptomatic Schizophrenia Amphetamione psychosis
Alcoholic hallucinosis
Auditory Hallucinations (primary)
Dellusions(secondary)
Toxic Psychosis due to hallucinogens
Mescaline, lysergic acid and psilocylin
Differentiable in color and shape constancy.
Paranoid psychosis
Delusions
Development of sensitive, suspicious and rigid personality.
Jealousy and erotomania
Cycloid Psychoses
Manic –depressive Illness
The excited pole and a inhibited depressesed pole
8. Amphetamines and cocaine
L-Dopa -increases the levels of dopamine
Neuroleptic drugs (chlorpromazine) ?Immediately on Hallucinations
?Weeks to affect the behaviour of the patients. Dopamine hypothesis is unable to explain this delay.
Chlorpromazine?little/no difference to 30%
9.Chlorpromazine - an anti-psychotic drug
Clozapine – To treat the Negative Symptoms
Both dopamine and serotonin are
probably involved, but the precise role played by each is unclear.
10.Acute Schizophrenia ( Type 1) + symptoms
? May make Complete recovery.
Chronic Schizophrenia
?Patient progressively may get disabled
10% - Rapid & Permanent Deterioration
35% - Mild & Persistent Symptoms
35% - Appear Cure but have relapse of Illness
20% - Apparently cure and stable
11.Physical
Drug Screening
Test for organic illness
Psycho- Social
History
Asses Home environment
12.Acute Phase
Long term Treatment
13.Pre Morbid:
Family History
Schizoid Personality
Home environment
Poor Previous Work Record
Illness
Gradual Onset
Absence of Prominent Affective Symptoms
Pronounced Negative Symptoms
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